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As acting the natural product Lastevolution

Lastevolution is a supplement that has proven very effective in rebalancing the decline of female sexual desire and encourage female ejaculation and libido by increasing the sexual satisfaction and pleasure.

We invite you to explore the information that you will find in these pages with the sincere hope that we can help eliminate your problem of lack of sexual desire by putting conditions to experience what few women know: female ejaculation once again and regain the pleasure and intimacy of a satisfying sex life.

Credible evidence between different cultures that the female prostate and female ejaculation were discovered, described and then forgotten over the last 2,000 years. Given that all women have a prostate gland, all probably produce ejaculate, even if you are not aware of it. The use of Lastevolution promotes a better response from the female prostate gland to stimulate and therefore improves the ability to achieve ejaculation. The female may produce prostate fluid (when a woman is sexually aroused) from 10 ml. to 60 ml. of liquid.

With “G-Crest” defines the “condition” of the enlarged prostate women during sexual arousal. All women can achieve this task and it is likely that all have at least a small amount of prostate fluid that leaks out, mixing with other fluids that are present in high amounts.

Lastevolution has an effect on the cardiovascular system, central nervous system, and breathing. Biological and chemical process that leads to the excitation are caused by nitrogen oxide which has the function of the nervous impulse signal that ease and relaxation of muscles and therefore the blood supply.

The active ingredients contained in Lastevolution promote release of nitrous oxide and it is for this reason that are also applications for the treatment of sexual problems. Not adverse outcomes were found and not known contraindicati-
ons.

The supplementary contribution of Lastevolution components provides an amount of chondrocytes of raw material to balance the difference between degradation and tissue renovation, lending support to a rapid recovery.

As acting the natural product Lastevolution

Both studies were carried out security tasks on Lastevoluti-
on components and on the basis of the results obtained can be evaluated as a naturally derived raw materials, innovative and able to assist in activities closely related to the sexual sphere. These active principles could play a key role in a supplement as Lastevolution.

Lastevolution use helps the sex glands that, with the passing of years, require a more significant amount of vitamins and procure from other tissues that would be exposed to diseases, increasing the use of zinc, magnesium, copper and potassium which are essential ingredients for the functioning of the seminal fluid.

The amino acid L-Histidine as contained in Lastevolution, plays a key role in sexual activity during ejaculation and orgasm. The body uses Histidine to produce histamine that is responsible for ejaculation. Men and women who have trouble achieving orgasm can be favored by supplementati-
on of Histidine, that is also a vasodilator that causes blood flow to sexual organs.
This amino acid is responsible, along with vitamin B3 (niacin) and B6 (pyridoxine) of the synthesis of histamine, which improves sexual activity and pleasure.

Female ejaculation comes from periurethral glands. These glands saturate it with the fluid during intercourse and the bulge of tissue can be heard from the vaginal wall. The liquid that emerges is composed also by glucose (a sugar) and fructose as the one present in Lastevolution formula.

Orgasmic gushing: where does the fluid come from and how is it produced

Women produce three sexual fluids: lubricity by means of exudation, fluid through the vaginal mucosa and mucus through vestibular glands; female ejaculation by paraurethral glands and gushing.

Orthodox Western medicine and Physiology have not yet issued an objective description or explanation the third, gushing.

We propose that it be a filtration of plasma produced by a process known as exudation. This is an additional application of oozing after vaginal lubrication.
Our thinking is that the fluid released by a Gush from the ventral wall of the vagina due to an increased surface area of the dilated arteriole and mucosa during the sex flush that sweeps over the vagina, stimulating veins pressurized and compression caused by muscle contraction during sexual climax and orgasm.

As acting the natural product Lastevolution

Introduction

Some cultures recognize three female sexual fluids of women including the written Tao, Zen and other Micronesian islands Trukese cultures described in works cited by Sevely. Western orthodox medicine recognizes two of these sexual fluids. The first is the lubrication and is due to the secretion of vulval vestibule glands (Bartholin’s glands) and exudation of fluid through the mucous membrane of the vagina. The second, called female ejaculation, consists in the secretion produced by the female prostate (gland Shene) and involves the paraurethral duct and the urethra until vulva featuring climax of some women. The third fluid is known in some areas as gushing.

Until more detailed discussion has developed of the G-spot by Grafenberg and “erotic zone” on the front wall of the vagina, a reasonable understanding had not yet been developed on the anatomy and physiology of paraurethral and female ejaculation. Significant advances have been made including detailed histological examination of the glands and their biochemical functions for the production of such components as chemical signals and prostate specific antigen. However, the confusion still exists when the paraurethral glands and female ejaculation are used in order to explain all the fluids expelled during female orgasm. The POV or female ejaculation is a separate phenomenon.

The POV or female ejaculation is described as a watery liquid, little or no smell and taste colored residuez. The quantity produced can vary from a few drops to 10 milliliters or non-specific. The issue can happen only once during session coital or can repeat with a series of climaxes. When are issued large amounts of fluid, the woman can accuse symptoms of dehydration. The fluid may be jettisoned as Gush that may be propelled forward with a Jet or can simply scroll without specific impulses. In the same way that the orgasm, ejaculation does not appear in all women and all those who succeed in this drive does not always manage to ejaculate because it is an irregular and unpredictable biological event.

In the same way that the orgasm, ejaculation does not appear in all our research allow us to say that the fluid released during female ejaculation is a filtrate of plasma produced by a mechanism known as the exudation. The fluid that is created to gush, out from the abdominal wall of the vagina where we find a rich vascular network, a dense venous Plexus, soft connective tissue and permeable epithelial membrane. The flow model has described various forms of POV as results of a sudden increase of intercellular liquid or pressure that is likely to create a wave of response due to vasodilatation reinforced by muscular contractions.

As acting the natural product Lastevolution

The research could solve some of the problems that have hindered until now acceptance and understanding of the phenomenon of of gushing. There is no external genital organ known as capable of producing fluid “on request” and in large quantities. So small glands such as paraurethral are not able. If instead of “on demand”, it was produced over a period of time, it should be stored somewhere as a vesicle or bladder but none of this has been found by anatomists. No ducts has ever been found empty inside the vagina but most descriptions make it understand that seems to originate from the ventral wall of the vagina apparently associated with stimulation of the G spot (female prostate). Some observers, however, believe that the fluid is produced from the urethra and sent directly into the vestibule.

Our research addresses the problem of the absence of a liquid storage container, believing that the fluid is produced “on demand”. The question “what organ would be capable of such fluid production?” is resolved in the description below, of arteriolar, capillary and epithelial exchange mechanisms of the vagina.

Production sources and expulsion of fluid

The model of gushing derived by transudation is expanded under three headings:

  • Source of fluid (Anatomy)
  • Production of fluid (Physiology)
    • 1. The surface area
    • 2. Impulse that causes the event (attendance, leakage, compression)
    • 3. Permeability
  • Fluid ejection (sexual climax).

The vaginal wall is composed of three main layers: mucosa, muscle and adventitious. The mucosa consists of a lamina propria of loose connective tissue and a very permeable non keratinized epithelium and scales. Both the adventitious that muscle and mucosa are composed of a large number of elastic fibers. The mucosa has many parts nerve terminals and also muscular. The latter would be significant roles in regulating vascular and sensitive control and pressure release.

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The mucosa has two longitudinal ridges on the surface epithelial medians, one front and one rear. Numerous bilateral roughness traverse extend from these vaginal “columns”. They are separated by grooves and variable depth. Many anecdotal studies on female gushing refer to these rugae on the ventral vaginal column.

The sulci were previously described as open ducts on the walls of the vagina. These apparent ducts were small recesses in the mucosa (gaps). Gaps in addition to the area of the flow in the lumen and is expected, not to be connected, with the paraurethral glands like ducts, surrounded by secretory cells in the urethra confluent. It should be pointed out that the Central wall is was so wrinkled that it was two or three layers of the Central wall and that there were many more rugae in the distal compared with the proximal vagina. Were described very small pores that oil and unload profusely during the sexual act. Should not confuse this with female ejaculation; should give properly female prostate through the urethra with expulsion.

The lamina propria consists of loose connective tissue also called connective tissue areolar. Like a collapsed sponge, this tissue has countless potential spaces able to grow and expand with the fluid. The wall of the vagina is rich in the arteries that are able to dilate if stimulated; consequently, the lamina propria becomes edematous. The veins and lymph that discharge vaginal wall are organized like networks including the venous Plexus and lymph plexus. The result is a thickening of vaginal wall that favors the Orgasmic platform that provides additional stimulation to the penis during coitus.

More blood arrives at the wall of vagina internal iliac arteries that have different ramifications. Noteworthy are the ramifications that come from the uterine arteries and internal uterine pudenda. The vaginal arteries anastomose to form a longitudinal median azygos artery on the ventral surface and another on the dorsal surface of the vagina, feeding the mucosaa.
Vaginal artery enlargement during stimulation and filling of veins Plexus cause mucosal edema and transudation for lubrication. Further mutations of exudation which lead to orgasm can trigger even female ejaculation.

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Exudation

Transudation is the filtration of water, electrolytes and some proteins from plasma, across a membrane, usually a serous membrane. The vaginal mucosa usually has no transudation without the contribution of a membrane. Important membranes are found in the pericardium, pleura and peritoneum where a thin film composed of active fluid lubricant, surfactant agent between the two opposing membranes.
The fluid that spreads on the mucous membrane of the vagina during the vasocongestion also provides the possibility to modify the friction during sexual activity, especially in combination with the mucus secreted from the cervix and cervical from most vestibular glands.

The glycogen from epithelial cell provides additional changes to the characteristics of the fluid. At rest, a small amount of fluid is the mucosal surface in order to maintain hydration. Upon awakening the lamina propria becomes edematous and substantial amounts are spread on the surface providing sufficient lubrication. During the climax, an additional increase in the dissemination of clear liquid can flow into the vagina and even gushing of the vagina.

When the feeder arteries and arterioles dilate, the dilated spaces of tissue of lamina propria are filled with fluid. When the capillary pressure increases around the serous membrane, exudation rate across the membrane increases in in direct proportion to the increase in pressure. From this it can be deduced that an increase in hydrostatic pressure in the interstices during the climax would lead to a predictable and measurable increase in transudation into the vagina during orgasm.

The speed with which this fluid can deteriorate and the significant amount of fluid that can be involved can be demonstrated with acute pulmonary edema: this event, such as the blocking of pleural lymphatic drainage, reduces cardiac pump or a severe drop in plasma colloid pressure, can result in transudation of one to two litres of fluid into the lungs in 30 minutes, leading to death.

Production of fluid

The chemical composition of the liquid due to female ejaculation may be compared with that of serum, plasma and transudate resumed from the same subject under other conditions. The vaginal fluid produced by exudation at rest is a modified solution plasma. Compared with plasma, vaginal transudate contains: high amounts of potassium, sodium, low amount of high amounts of urea, typical plasma proteins, albumins and globulins plasma, acetic acid, lactic acid and other various volatile fatty acids.

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Under conditions of sexual arousal, increases the rate of production of fluid and the composition is not changed; the sodium and water reabsorption mechanisms are exceeded. Such changes occur when the sweat or the salivary secretion increases from baseline to the maximum and the mechanism that can normally reabsorb sodium, potassium excreted or absorb the solvent has little time to act; as well as the scale increases, the proportion of sweat or saliva which are essentially filtered plasma, they constitute the largest portion of secretion.

On the basis of the chemical composition of the exudation and its change with increasing flow during the activity, we can say that the fluid produced during ejaculation exhibits a great deal of change. As the production of fluid reaches a peak, it could overwhelm the capacity of the mucosa to change its composition and consequently is approaching to that of the serum.
We need to describe a task that can conduct a large amount of fluid in a membrane in a short time. In biological systems, the increases in the spread are made by an increase in the surface area where the flow; a driving force as, in this case, is the hydrostatic pressure gradient that increases the diffusion coefficient and altering the permeability of the membrane.

Increase on the surface area where is the flow

The vaginal wall has three features that have the potential to increase the capabilities of the area. Deep waves reside on the interface between the epithelium and the lamina propria. This feature is shared with urothelium that allows mucus stretch until the surface epithelial luminal and concomitantly decreases the distance through which the fluid can spread. The folds of the abdominal wall encloses the excess area that becomes available, flattening the cavity as a result of movements, muscle contraction or development of localized edema. The “curtains” or bulging proximal of the vagina are the reason why the cervix expands its surface and flattens the mucosa.

Increase stimulus

Sometimes the oozing through the permeable membrane. It is basically composed of water contained in the electrolytic solution or plasma-induced mass flowing when vasoconstriction. The pressure gradient can increase with the increased pressure of the artery or by increasing the rate of inflow of blood; the increase in venous pressure for the spillage of blood or lymph; squeezing the tissue spaces as happens during muscle contraction, smooth muscle scheletrale or cell epithelial dotted lamina propria.

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Increased venous pressure increases transudation at serosal membranes such as the pleura and pericardium; so it can be predicted to be a major contributor in the vagina also. Surely the venous pressure increases during the vasocongestion which causes an erection. Thus transudation sufficient for lubrication accompanies erection. But what causes the additional flow called gushing?

There should be sudden increases in pressure in interstitial tissue. The hydrostatic pressure is the result of an imbalance between inflow and outflow, then a sudden influx that has not been combined with increased outflow would lead to a significant hydrostatic pressure in the tissue spaces, increasing the flow through the walls of the mucosa. In the pericardium this mechanism is tested systematically by elevating myocardial venous pressure and measuring increases in epicardial transudation / pericardial effusion.

What can cause a sudden increase in pressure or rate of inflow? The hydrostatic pressure of the feeder arteries is determined by the systolic blood pressure. During orgasm the pressure increases and the woman systolic pressure doubles during each orgasm. This high blood pressure increases the driving force that activates the transudation  mechanism.

Inflow is provided through the median longitudinal vaginal arteries and arterioles beyond mucosa. A wave of hyper polarization as that which causes the release of nitric oxide can temporarily decrease arteriolar tone and increase in capillary flow feeding the interstitial fluid of the vaginal wall. Nitric oxide has the characteristic of having a short activity before being metabolized, so a gushing jet of fluid can sometimes be released. In the woman’s vagina have been identified nitrergic neurons associated with blood vessels.

The skin flush that usually characterizes orgasm is characterized by a sudden wave of peripheral vasodilatation (reminiscent of the menopause hot flashes). Female ejaculation can be a localized result due to vaginal arousal? These are possible and vasodilating effects mediated by the sympathetic cholinergic system-leaving the anterior hypothalamus, as happens during sudden arousal or sweating during a febrile or inhibition of vasoconstriction by the sympathetic system from the hypothalamus.

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Are also possible activities motor parasympathetic fibers. Rapids and copious amounts of fluid present in the stomach in response to vaso-vagal reflex. In this gastric reflex, sensory information is sent from the vagus nerve from the brainstem, and from the top of the stomach in the vagus nerve.
The reflexes starts with the distension of the stomach mucosa and tactile stimulation of the surface of the mucous membrane of the stomach. This gastric secretion can begin with the signals sent by the brain, especially the limbic system.

These components have a remarkable similarity reflexes with the phenomenon that affects the vagina except those vaginal reflexes would act via sacral pathways. Although the central origin of the innervation has not been clarified, the peripheral synapses have been found to be not atropine sensitive,so that the answers are likely mediated by vasoactive intestinal polypeptide (VIP).

The reduction of flow also increases the interstitial pressure and exudation. Tonic contractions of the vagina wall can easily close the venous Plexus and avoid the release of blood as it does for the erection of the penis. Studying the secretion of oxytocin during orgasm, there has been a wide fluctuation of blood flow during each subsequent contractile function of genital muscles.
Blood flow was monitored using wave photos. The mechanism refers to that here is observed readily in the lung where, if venous or lymphatic drainage fails causing even slight, increase in interstitial pressure, the intracellular fluid passes through the space of the alveolar area causing pulmonary edema.

The serous membrane of the pleura demonstrates such a high fluid flow if requested a significant backpressure. In erectile tissues venous outflow can be blocked by compression of veins, or by vasoconstriction of vessels draining the tissue. The stimulus caused by colon and neuropeptid Y and blood vessels of the vagina may regulate vasoconstriction appropriate.

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In addition to disrupt the lymphatic vessels, the contractions of vaginal wall would squeeze the mucosal “sponge” This would further the interstitial pressure during the climax and will push the fluid out and into the lumen can be realized with a vaginal compression in an aroused subject.
During orgasm may be realized through smooth muscle contractions of the walls of the vagina or fibro muscular adhesions contractions of skeletal muscle as the bulbospongiosus.

The vaginal plethysmography used to monitor vessel congestion of vagina shows a slight decrease in the vasocongestion associated with contractions of pubococcy-
geus muscles if needed during the climax, and this is consistent with the fluid expelled during contractions.
An abundant amount of congestion, drops in the vase once it has been reached orgasm, also implies that significant amounts of liquid are sprayed out of the tissue by strong pelvic muscle contractions, leading to a decrease in tissue edema.

Increased permeability of the membrane

The speed of the fluid flow through a membrane greatly increases if the same thins or becomes more permeable. The vasodilatory peptide calcitonin gene-related (CGRP) occurred that contains neurons in vaginal mucosa with a specular role of capillary permeability regulator that can benaltered programmatically neural causing small changes in electrical potential in the membrane of capillary endothelial cells.

Water and electrolyte secretion from glands of the gastroin-
testinal tract are verified by activating the parasympathetic, resulting in hyperpolarization of the cell membrane and influx of chloride, water and flow drive electrolyte on the surface of luminal epithelial cells.
This mechanism is used to increase flow through cholinergically innervated gastric and salivary glands that can induce changes in 20-fpld rates quantities of fluid. It’s been used the membrane potential and the electrolyte concentration in the vagina to confirm the significant potassium transport mechanism where the flow is regulated through electrolytic concentrations in the vagina to confirm the significant potassium transport mechanism there i.e. electrolyte flux across the vaginal mucosa is regulated, not passive (at least, not at rest).
The question of how the electrolytic is tuned flow seems to have an answer in Immunohistochemistry which finds containing nerves calcitonin gene-related peptide and nitric oxide, VIP (suprachiasmatic nuclei) and neuropeptide Y (NPY) associated with blood vessels in the human vagina.

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Ejecting fluid

Some research has already reported on the implication of electrolyte activity such as exuding neurogenic which provides in quantities above the liquid due to hydrostatic pressure changes as described previously. The drip filtered through the intercellular spaces of vaginal epithelium acts by saturating the Na + reabsorption and limited capacity of cell transfer. Thinning of the epithelium may facilitate the spread of the fluid.
In this way, the increased surface area increases the imbalance between the supply and the blood flow and increased mucosal permeability, would each add to the delivery of a pulse of fluid at orgasm.

Why the ventral wall?

There are several explanations for the the vaginal ventral column may be more involved in ejaculation that the dorsal column. Can pump several times as the dorsal wall. The urethra is embedded in the abdominal wall. The erection of the corpus spongiosum surrounding the urethra can cause swelling of the urethral tissue in the vagina, increasing visibility and palpability of the abdominal wall. The ventral wall also helps to anchor the external urethral sphincter.

This anchor would tend to stabilize the ventral column in such a way that the contractions of adjacent muscles would squeeze it rather than passively drag it. The female prostate tissue is distributed dorsally in the wall of the urethra into the vagina and greater sensitivity, known as the G-spot, is associated with it. This sensitivity probably encourages humans to choose the coital position that increases directly the ventral wall of the vagina, and further increases the likelihood that it gushing mechanisms will be activated.

Extrapolation of a considerable volume of fluid during female ejaculation

The development of this study on female ejaculation was inspired by the attempts of some to explain the large amount of fluid expelled by some women during orgasm. The female paraurethral prostate glands may not be able to produce large volumes of fluid required as using the apocrine and merocrine functionality. Prior to secretion production would require a storage.

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Extrapolation of a considerable volume of fluid during female ejaculation

It is unlikely that a body enough to store large quantities of liquid has not been seen by anatomists for thousands of years. The only structure with suitable capacity and access to the outside is the urinary bladder but the description of experiences it forecloses the possibility given the chance by women to produce urine before and after reaching orgasm and also the highest concentration of PSA (prostate specific antigen) and the low concentration of urea of fluid. The only solution would be for an organ capable of producing large amounts of fluid “on request” and this research describes this well understood mechanism into the vagina.

Despite the ability of the vagina to produce large amounts of fluid on request, many episodes indicate that the fluid is often (but not always) seen out of the urethra and vagina. The same mechanism could also be present in the urethra? ... Perhaps. Transactional epithelium in the neck of the bladder, in the region known as trigone, is commonly replaced by an epithelium in women similar to that of the vagina but not in men.

Remain some gaps that need to be investigated on histological and physiological anatomy of the urethra. The recent model developed will provide a simple structure to use when studying the mechanism shown significant spill of liquid volumes in the form of a Gush from vagina might also explain the expulsion of a large amount of sexual fluid from the urethra during orgasm.
If appropriate microstructures have been identified, it will not be necessary to postulate a new mechanism for the watery secretion as the same mechanism has already been included. All that is needed is to check whether this mechanism can occur in the urethra. Investigation should explore the mucosa that lines the lacunae along the urethral walls, in regions which are not occupied by the prostate tissue.

Conclusions

LThe research that we have proposed is to understand how the female ejaculation is most likely the result of sexual arousal that makes wet vagina, increase mucosal blood flow and all accompanied by contractions of the vagina that increase the pressure in the venous plexus.
All of these factors increases the transudation through the mucosa. Transudation is probably enhanced or augmented by contractions and actually the liquid is pushed out from the lamina propria and the gap and sometimes fluids are pushed outside of the vagina with a strong pressure.